Sources of testosterone

NOTE:   One interesting fact about erythropoitin is that it is also called EPO, a name that may be more familiar to some of you.  Yes, that should bring the name Lance Armstrong to mind.  Lance Armstrong confessed to using EPO as a performance enhancing drug strictly for the purpose of raising his red blood cell counts for racing purposes.  Basically, he was doing a different and very dangerous kind of steroid.  Giving EPO to these racers leaves them vulnerable to dieing in their  sleep from the "sludging" of the blood that occurs.  It does turn them into superhumans however.

The second theory is similar and is known as "evolutionary neuroandrogenic (ENA) theory of male aggression". [79] [80] Testosterone and other androgens have evolved to masculinize a brain in order to be competitive even to the point of risking harm to the person and others. By doing so, individuals with masculinized brains as a result of pre-natal and adult life testosterone and androgens enhance their resource acquiring abilities in order to survive, attract and copulate with mates as much as possible. [79] The masculinization of the brain is not just mediated by testosterone levels at the adult stage, but also testosterone exposure in the womb as a fetus. Higher pre-natal testosterone indicated by a low digit ratio as well as adult testosterone levels increased risk of fouls or aggression among male players in a soccer game. [81] Studies have also found higher pre-natal testosterone or lower digit ratio to be correlated with higher aggression in males. [82] [83] [84] [85] [86]

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One human intervention using 200mg daily in men for 90 days (dose determined from Ayurvedic recommendations) failed to note any clinically significant toxicological symptoms when measuring standard toxixological biomarkers, but noted a small decrease in serum creatinine by % and increases in both Haemoglobin (no morphological changes of RBCs) and WBC count by % and 6% respectively. [8] A larger dose of 2g Shilajit (% fulvic acid) daily for 45 days in humans aged 16-30yrs did not note any significant toxicological signs in serum, but did not note any significant influence on haemoglobin. [12]

Transdermal patches (adhesive patches placed on the skin) may also be used to deliver a steady dose through the skin and into the bloodstream. Testosterone-containing creams and gels that are applied daily to the skin are also available, but absorption is inefficient (roughly 10%, varying between individuals) and these treatments tend to be more expensive. Individuals who are especially physically active and/or bathe often may not be good candidates, since the medication can be washed off and may take up to six hours to be fully absorbed. There is also the risk that an intimate partner or child may come in contact with the application site and inadvertently dose himself or herself; children and women are highly sensitive to testosterone and can suffer unintended masculinization and health effects, even from small doses. Injection is the most common method used by individuals administering AAS for non-medical purposes. [45]

Sources of testosterone

sources of testosterone

One human intervention using 200mg daily in men for 90 days (dose determined from Ayurvedic recommendations) failed to note any clinically significant toxicological symptoms when measuring standard toxixological biomarkers, but noted a small decrease in serum creatinine by % and increases in both Haemoglobin (no morphological changes of RBCs) and WBC count by % and 6% respectively. [8] A larger dose of 2g Shilajit (% fulvic acid) daily for 45 days in humans aged 16-30yrs did not note any significant toxicological signs in serum, but did not note any significant influence on haemoglobin. [12]

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